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[34] reported that when buthionine sulfoximine is used to inhibit the activity of GCS (which catalyzes the first step in GSH synthesis) a half life of 26 hours for cellular GSH is observed

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reported that H5N1 IAV infection in lung epithelial cells (A549 cells) decreased Cu/Zn superoxide dismutase (SOD1) expression, which could enhance ROS production, viral replication, and the inflammatory response 27